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Current and Emerging Treatment Options for Uterine Fibroids | Drugs | Springer Nature Link This model proposes that mGSH depletion due to its covalent binding to NAPQI acts as a first-hit, which is followed by a burst of mitochondrial ROS production that activates a second hit involving mitogen-activated protein kinases (MAPKs) that in turn amplifies the wave of mitochondrial ROS generation ( Figure 3 ) mg:5mg/5mg X 5pk https://media.springernature.com/m685/springer-static/image/art%3A10.1038%2Fs44298-025-00110-3/MediaObjects/44298_2025_110_Fig1_HTML.png Spot Patch
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