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To assay the ability of HPV-16 E7 Mut to interact with GSTP1 in vivo , control, HPV-16 E7-infected and HPV-16 E7 Mut-infected HaCaT cells were lysed and immunoprecipitation using an anti-HA antibody was performed
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A mechanism for the unchanged tissue glutathione content in riboflavin deficiency could be the increased biosynthesis of glutathione from its precursor amino acids as a compensatory action ( Reference Kaplowitz, Aw and Ookhtens 30 )
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